“Coolest Te Coolest” has an impeccable pedigree. He is a superb blend of the some of the greatest horses of all time in both the AQHA and ApHC industries.
His sire, “Te Coolest” needs no introduction to the appaloosa fan. He is a 3X World Champion, a US National Champion, and one of the industry’s leading sires.
His dam, “Cool Memories”, was a Reserve National Champion and a California State Champion during her show career. She is sired by one of the AQHA's leading broodmare sires; “Coolest”.
“Coolest Te Coolest” was shown to thirteen judges on the very tough California show circuit. He was Grand Champion or Reserve Grand Champion EVERY time shown. He placed in the top 5 at the National Championships as a yearling and again as an aged stallion.
Coolest Te Coolest stands 15.3 hands. And, as an added bonus, his offspring are turning heads as performance horses as well.
Re Coolest will be standing for $850 at Linn-Benton College, Albany, Oregon
The $850 stud fee will include the initial shipment of cooled semen, or 15 days of free mare care at the college.
Breeding season will be 02/01/2024 to 06/01/2024
Genetic Disorders explained
Hyperkalemic Periodic Paralysis Disease (HYPP) Description: Equine Hyperkalemic Periodic Paralysis Disease (HYPP) is a muscular disease caused by an inherited genetic mutation. HYPP has been traced back to one horse named Impressive and has the alternative name, Impressive Syndrome, named after this horse. Symptoms of HYPP may include muscle twitching, unpredictable paralysis attacks which can lead to sudden death, and respiratory noises. Severity of attacks varies from unnoticeable to collapse or sudden death. The cause of death is usually respiratory failure and/or cardiac arrest. HYPP is a dominant disorder meaning both homozygous positive (HH) and heterozygous (nH) horses will be affected. Only homozygous negative (nn) horses are not affected by HYPP. Because HYPP is dominant disorder, the effects of it can also be transposed to other breeds of horses when intermixing occurs. This makes the recognition of this disorder very important in preserving the inherited health of all horses.
Hereditary Equine Regional Dermal Asthenia (HERDA) Description: Also known as Hyperelastosis Cutis, HC is a genetic skin disease predominantly found in the American Quarter Horse. Researchers at Mississippi State University and Cornell University believe that the origin of this genetic disorder may be the Poco Bueno's sire line. The symptom of this disorder is a lack of adhesion within the layers of skin due to a genetic defect in the collagen that holds the skin in place. This defect causes the outer layer of skin to split or separate from the deeper layers, sometimes tearing off completely. Areas under the saddle seem to be most prone to these lesions, often leaving permanent scars and preventing the horse from being ridden. The disorder is recessive, which means that a horse must be homozygous positive or have two copies of the defective gene to suffer from the disease. Consequently, both the sire and the dam must possess at least one copy of the mutated gene in order for the offspring to be afflicted. Offspring born with one copy of the defective gene and one non-defective copy are considered a carrier and have a 50% chance of passing the defective gene on. Although there is no cure for the disorder, Animal Genetics has developed a simple DNA test to detect a single nucleotide polymorphism (SNP) mutation with a high degree of association to this disorder.
Glycogen Branching Enzyme Deficiency (GBED) Description: Glycogen Branching Enzyme Deficiency (GBED) is a fatal condition caused by the bodies' inability to properly store sugar. In a normal horse, the body stores sugar as energy by converting glucose to glycogen. This inherited disorder prevents the body from producing the enzyme needed to branch the glycogen structure, preventing the horse from being able to adequately store the sugars. This means that the horse will not be able to store enough energy to fuel important organs, such as the muscles and brain. Foals born which are affected by GBED suffer from a range of symptoms associated with this lack of fuel, such as low energy, weakness and difficulty rising. Other symptoms include low body temperature, contracted muscles, seizures, and sudden death. Unfortunately, GBED is always fatal; most affected foals will die before the age of 8 weeks. GBED often causes the foetus to be aborted in utero. Research suggests that as many as 3% of aborted Quarter Horse foals were homozygous for the GBED mutation. Studies show that the mutation responsible for GBED is carried by as many as 10% of Quarter Horse, Paint Horse breeds and related breeds. GBED is an autosomal recessive trait, meaning a foal can only be affected if the foal inherits the disease from both parents. Horses that are carriers of the GBED have one copy of the mutation but do not have any symptoms associated with the disorder. This makes DNA testing important to screen for carriers and prevent this fatal condition. The mutation which causes this disease, has been identified by Dr. Stephanie Valberg and Dr. James Mickelson of the University of Minnesota, and is licensed for diagnostic use by Animal Genetics Inc (USA). Breeds Affected: Quarter Horse, any horse with quarter horse blood.
Polysaccharide Storage Myopathy (PSSM) Description: Polysaccharide Storage Myopathy (PSSM1) is a dominant autosomal hereditary condition that can cause a genetic form of tying-up with muscle damage and inability to move. One form of PSSM1 is in part a result of a single base pair substitution in GYS1 gene, thereby changing the amino acid sequence of the glycogen synthase enzyme. At least 20 breeds have been identified with Type 1 PSSM. The prevalence of the GYS1 mutation in Belgians is as much as 50% of and 8% of the Quarter Horse-related breeds. Some horses make and store abnormal muscle glycogen and cannot tolerate dietary starches and sugars. Horses with PSSM1 can be maintained with low-starch and low-sugar rations and precise exercise protocols. In some horses symptoms may begin by 2 to 3 years of age while others can remain subclinical. Clinical signs can include skin twitching, stiffness, firm painful muscles, sweating, weakness, and reluctance to move with light exercise. Occasionally gait abnormalities, mild colic and muscle wasting may also occur. In may cases horse that have tested positive have had no history of 'tying-up' or other symptoms associated with PSSM1. Research conducted at Animal Genetics has identified several additional mutations associated with PSSM1. These DNA mutations form a haplotype that allow us to identify horses with PSSM1. Further ongoing research may provide us with a more comprehensive assay for PSSM and better determine the severity of the disorder in all horses. An additional genetic mutation in RYR1 gene (MH) influences PSSM and increase the severity of the symptoms of PSSM1 in Quarter Horses and related breeds. The combination of PSSM and MH genetic tests are available at Animal Genetics. Not all cases of tying up are caused by the PSSM1 mutations currently being looked at. A horse that tests N/N for PSSM1 but exhibits signs of tying-up or muscle pain may be suffering from an another muscle disorder. Please contact us with any examples of this so we can include these animals in further studies. In many cases horses that test positive for these mutations will exhibit only minor problems or may never exhibit any noticeable problems at all. Again, please contact us and let us know so we can add these animals to our current research on PSSM1.
Malignant Hyperthermia (MH) Description: Malignant Hyperthermia or MH is a genetic muscle disorder that affects Quarter Horses and related breeds. Horses with the MH mutation may not show any physical signs of the disorder until triggered by exposure to anesthesia or extreme exercise or stress. Symptoms can include high temperature, increased heart rate, high blood pressure, sweating, acidosis, and muscle rigidity. Symptoms develop rapidly, and if not treated quickly, this condition can be fatal. MH is inherited as an autosomal dominant trait, so the disorder can be passed on even if only one parent has the defective gene. The mutation can be present along with PSSM and if a horse also has PSSM, the symptoms associated with MH can be more severe. Therefore, testing for both PSSM and MH is recommended for Quarter Horse breeds. Although this condition is rare, testing for MH is recommended in case a horse must undergo anesthesia. Horses that are known to have the MH mutation can be given medication prior to administering anesthesia to help reduce the severity of the symptoms.